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Hormonal factors in the control of the browning of white adipose tissue

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Hu, Jiamiao and Christian, Mark (2017) Hormonal factors in the control of the browning of white adipose tissue. Hormone Molecular Biology and Clinical Investigation, 31 (1). 20170017. doi:10.1515/hmbci-2017-0017 ISSN 1868-1883.

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Official URL: http://doi.org/10.1515/hmbci-2017-0017

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Abstract

Adipose tissue has been historically classified into anabolic white adipose tissue (WAT) and catabolic brown adipose tissue (BAT). Recent studies have revealed the plasticity of WAT, where white adipocytes can be induced into ‘brown-like’ heat-producing adipocytes (BRITE or beige adipocytes). Recruiting and activating BRITE adipocytes in WAT (so-called ‘browning’) is believed to provide new avenues for the treatment of obesity-related diseases. A number of hormonal factors have been found to regulate BRITE adipose development and activity through autocrine, paracrine and systemic mechanisms. In this mini-review we will discuss the impact of these factors on the browning process, especially those hormonal factors identified with direct effects on white adipocytes.

Item Type: Journal Article
Subjects: Q Science > QP Physiology
Divisions: Faculty of Science, Engineering and Medicine > Medicine > Warwick Medical School > Biomedical Sciences > Cell & Developmental Biology
Faculty of Science, Engineering and Medicine > Medicine > Warwick Medical School
Library of Congress Subject Headings (LCSH): Adipose tissues, Fat cells, Obesity -- Treatment, Metabolism -- Regulation
Journal or Publication Title: Hormone Molecular Biology and Clinical Investigation
Publisher: Walter de Gruyter
ISSN: 1868-1883
Official Date: 21 July 2017
Dates:
DateEvent
21 July 2017Published
11 May 2017Accepted
Volume: 31
Number: 1
Article Number: 20170017
DOI: 10.1515/hmbci-2017-0017
Status: Peer Reviewed
Publication Status: Published
Access rights to Published version: Restricted or Subscription Access
Date of first compliant deposit: 31 August 2017
Date of first compliant Open Access: 21 July 2018

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